FISIOPATOLOGIA DEL RCIU PDF

Malashakar You will only be able to see the first 20 seconds. Los autores desean agradecer al Dr. The action of severe maternal hyperglycemia depletes the fetal pancreas, which loses the ability to produce insulin at the end of pregnancy. Figure 2 — Mean weight of the placentas from the control groups, the group submitted to dietary restriction from the 1st day and the group submitted to dietary restriction after the 3rd day of pregnancy, as determined on the 18th and 21st days of pregnancy.

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Arashirn Figure 6 — Placentas of rats receiving the diet ad libitum A and of rats submitted to protein-calorie restriction from the 1st day B and after the 3rd day of pregnancy Cobtained on the 18th day of pregnancy — in Hematoxylin-Eosin and Periodic Acid Schiff.

The placentas were separated from their membranes, weighed and processed for histopathological study morphology and histochemistry. Between [uly and April Doppler ultrasound of the uterine arteries was performed in pregnant women participating in the study and uterine arteries indexes and velocities were calculated; media were compared and RCIU diagnosis validity tests were performed. After removal of the uterus and separation of the placenta, newborn rats were weighed and classified as small SGAappropriate AGA and large LGA for gestational age of 18 and 21 days as compared to the mean and Standard for the control group.

Un cuerno uterino es la bocina y otro caso es el cuerno fisippatologia control. Fisiopatologia rciu pdf file An evaluation of the efficacy of Doppler flow velocity waveform analysis as a screening test in pregnancy.

Doppler velocimetry of the uterine artery as a screening test for gestational hypertension. Histopathologic examination of the fetal pancreases did not shown the effects of maternal malnutrition Table 1 and Figures 4 and 5. How to cite this article. Br J Obstet Gynaecol. Pdf the antiphospholipids syndrome saf, is a disease of the immune system, in which antibodies with apparent specificity by phospholipids with negative electrical charge exist.

The lack of insulin production by the fetal pancreas at the end of pregnancy does not permit the removal of glycogen stores from the placenta and the fetus does not grow in an adequate manner. Figure 3 — Percentage of small for gestational age newborn rats from the control group, the group submitted to dietary restriction from the 1st day and the group submitted to dietary restriction after the rci day of pregnancy, as determined on the 18th and 21st days of pregnancy.

This is probably due to the vicarious action of the placenta occurring with its increased weight Figures 1 and 2. Basic studies and clinical implications. In the groups submitted to dietary restriction from the 1st day of pregnancy, fetal development was normal up to the 18th day because of the vicarious action of the placenta and of the glycogen stores. At the end of pregnancy, the weight of the placentas of rats submitted to dietary restriction from the 1st day of pregnancy was the same as the weight of control rats and higher than that of the group submitted to dietary restriction after the 3rd day Figure 2.

Please recommend JoVE to your librarian. Reto para el ginecoobstetra. Placental glycogen stores also decreased after fiaiopatologia 18th day in the control groups and in the groups submitted to dietary restriction from the 1st day of pregnancy.

Early fciu changes in hemodynamics and volume homeostasis are consecutive adjustments triggered by a primary fall in systemic vascular tone. An unexpected error occurred. Doppler investigation of uteroplacental blood flow resistance in the second trimester: The 41 rats produced young; the mean number of newborns per rat was Get cutting-edge science videos from J o VE sent straight to your inbox every month. Fill out the form below to receive a free trial or learn more about access:.

We recommend downloading the newest version of Flash here, but we support all versions 10 and above. Experimental research carried out in Botucatu Brazil has demonstrated that fetuses with IUGR are obtained using models of protein-calorie malnutrition, of maternal arterial hypertension and of severe maternal diabetes. RCIU: by sofia cordoba on Prezi Data concerning newborn weight and placental weight were analyzed by fully randomized factorial analysis of variance.

In the pancreas of newborn rats with severe diabetes the islets are large but have no insulin-positive cells and therefore their insulin production is depleted. The continuation of maternal malnutrition up to the 21st day was associated with decreased fetal weight and increased incidence of intrauterine growth retardation even though placental weight rcik maintained and glycogen stores evolved normally.

Mean gestational age for the IUGR and control groups were respectively The fisioptaologia of pregnancy up to the 21st day shows that protein-calorie restriction is a model for IUGR Figure 3 associated with decreased placental weight Figure 2confirming previous reports.

Int J Mol Sci. Dietary restriction after the 3rd day of pregnancy led to low placental glycogen concentrations on the 18th day and disappearance on the 21st day. Arch Latinoamer Nutr b;33 1: The general objective of the present investigation was to study the physiopathology of intrauterine growth retardation in fetuses in pregnant rats submitted to protein-calorie malnutrition, on the basis of evolution of the placenta and of the fetal pancreas.

Course of maternal blood glucose levels and its repercussions on the blood glucose levels and pancreas of newborn pups. The nature of the etiologic agent, the time when the attack occurred during. Francesca Russo, la Sra. When pregnancy was resolved on the 21st day, fetal weight was decreased in the group of pregnant rats submitted to dietary restriction, and more intensely in the group in which restriction was started after the 3rd day Figure 1.

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